详情页 - 首都医科大学宣武医院知识库

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作者：

Hai-Tao LiWeiWang[1,1,2,3,4,5] * ; Ting-ting Hou[1] ; Hua-RU-Yuan Tian[1] ; Qiaoqi Wu[1,2,3,4,5]

; Yi-Ping Wei[1,2,3,4,5]

; Ling-Zhi Xu[1,2,3,4,5]

; Xiu Wang[1] ; Jianping Jia[2,3,4,5,6]

;

机构： [1]Xuan Wu Hospital, Capital Medical University,Beijing, China [2]Center of Alzheimer’s Disease, Beijing Institute for BrainDisorders, Beijing, China [3]Clinical Center for Neurodegenerative Diseaseand Memory Impairment, Capital Medical University, Beijing, China [4]KeyLaboratory of Neurodegenerative Diseases, Ministry of Education, Beijing,China [5]Beijing Key Laboratory of Geriatric Cognitive Disorders, Beijing,China [6]Innovation Center for Neurological Disorders, Department ofNeurology, Xuan Wu Hospital, Capital Medical University, Beijing, China.

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出版当年[2017]版：

大类 | 1 区

医学

小类 | 1 区临床神经病学

最新[2025]版：

大类 | 1 区

医学

小类 | 1 区临床神经病学

第一作者：

第一作者机构： [1]Xuan Wu Hospital, Capital Medical University,Beijing, China [2]Center of Alzheimer’s Disease, Beijing Institute for BrainDisorders, Beijing, China [3]Clinical Center for Neurodegenerative Diseaseand Memory Impairment, Capital Medical University, Beijing, China [4]KeyLaboratory of Neurodegenerative Diseases, Ministry of Education, Beijing,China [5]Beijing Key Laboratory of Geriatric Cognitive Disorders, Beijing,China

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MLA：

HONOKIOL ALLEVIATES COGNITIVE DEFICITS OF AD (PS1 V97L ) TRANSGENIC MICE BY ACTIVATING MITOCHONDRIAL SIRT3

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