详情页 - 首都医科大学宣武医院知识库

当前位置：首页 > 详情页

Pharmacological effects of ginsenoside Rg1 in neuropsychopharmacology

文献详情

资源类型：

作者：

机构： [1]State Key Laboratory of Bioactive Substances and Functions of Natural Medicines, Institute of Materia Medica,Neuroscience Center, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing 100050,China [2]Department of Pharmacy, Xuanwu Hospital of Capital Medical University, Beijing 100053, China [3]Hunan University of Chinese Medicine, Changsha 410208, China

出处：

关键词： ginsenoside Rg1 neuropsychopharmacology multiple targets

基金：

语种：

第一作者：

第一作者机构： [1]State Key Laboratory of Bioactive Substances and Functions of Natural Medicines, Institute of Materia Medica,Neuroscience Center, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing 100050,China [2]Department of Pharmacy, Xuanwu Hospital of Capital Medical University, Beijing 100053, China

通讯作者：

通讯机构： [1]State Key Laboratory of Bioactive Substances and Functions of Natural Medicines, Institute of Materia Medica,Neuroscience Center, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing 100050,China [3]Hunan University of Chinese Medicine, Changsha 410208, China

推荐引用方式(GB/T 7714)：

APA：

MLA：

相关文献

[1]Ginsenoside Rg1 prevent and treat inflammatory diseases: A review [2]人参皂苷Rg1对β淀粉样蛋白诱导细胞凋亡的影响 [3]Ginsenoside Rg1 attenuates beta-amyloid-induced apoptosis in mutant PS1 M146L cells [4]Ginsenoside Rg1 promotes proliferation and neurotrophin expression of olfactory ensheathing cells [5]Ginsenoside Rg1 Inhibits Microglia Pyroptosis Induced by Lipopolysaccharide Through Regulating STAT3 Signaling. [6]UPLC-MS-MS同时测定中药活血益气方KLW中三七皂苷R1、人参皂苷Rg1、人参皂苷Rb1的含量 [7]HPLC法测定抗瘤丸中三七皂苷R1及人参皂苷Rg1、Rb1的含量 [8]Ginsenoside Rg1 Inhibits Microglia Pyroptosis Induced by Lipopolysaccharide Through Regulating STAT3 Signaling. [9]Ginsenoside Rg1 protects mice against streptozotocin-induced type 1 diabetic by modulating the NLRP3 and Keap1/Nrf2/HO-1 pathways. [10]Ginsenoside Rg1 Inhibits Microglia Pyroptosis Induced by Lipopolysaccharide Through Regulating STAT3 Signaling.