Introduction Cerebral air embolism (CAE) is an uncommon cause of stroke, typically arising when intravascular air enters the cerebral circulation via trauma or iatrogenic events (e.g., catheter insertion) (1). Although rare, CAE carries a high risk of severe neurological injury, with significant morbidity and mortality (1). Common risk factors include central venous catheter manipulation, endovascular or cardiothoracic interventions, laparoscopic surgery, and neurosurgical procedures performed in the sitting position (2). Clinically, CAE can be challenging to recognize, as patients often develop non-specific neurologic symptoms (altered consciousness, seizures, or acute stroke-like deficits) in close temporal relation to a precipitating procedure (2). Given its often abrupt presentation and potential to rapidly deteriorate, prompt diagnosis is critical.